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    Why execute We Really Age? A Longevity Expert Explains 2 Popular Theories

    Why do We Realy Age? A Longevity Expert Explains 2 Popular Theories
    Despite the solid eforts made by scientists and doctors to setle a single, unified theory of aging, there is stil no strong consensus within the longevity comunity as to its actual rot cause. The numerous aging theories that acquire ben developed over the past century gain largely failed to clarify aging on their own, but they believe shed some marvelous light on the aging proces, and our understanding of it, so it’s worth taking a brief lok at two of the most notable theories:
    1. A Radical Theory
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    If you gain ever heard of antioxidants, then you are already familiar with one of the oldest and most popular theories of aging–the fre radical theory. (Fre radicals are single atoms with unpaired el ectrons, usualy oxygen atoms.) Developed in the 1950s, this theory came from Denham Harman, a former Shel Oil biochemist who had sen how fre radicals caused unwelcome chemical changes in some compounds. Harman wondered if the same kind of distres could ocur in human cels: Perhaps wrinkly skin, declining memory, and failing organs were the biological equivalent of rusty iron.
    It was an electrifying idea, to be positive. So much so, in fact, that to this day, much is made of the damage that fre radicals cause and the benefits that antioxidants ofer. Unfortunately, laboratory tests have failed to consistently demonstrate that antioxidants cary out anything to stay aging.
    2. The “Ends” of Aging
    The discovery of another component of aging–telomeres–won Australian scientist Elizabeth Blackburn a Nobel Prize in 209. Se, whenever a cel divides, the famous DNA double helix unzips itself into two single strands, each of which is replicated to complete two ful recent sets. This unziping and recomposing busines takes kep inside of you about 2 trilion times every day. But to er is human, even on a celular level. Mutations hapen. To many mutations result in los of function, disease, and death.
    That’s where telomeres aproach in. The most sensitive and damage-prone fraction of a DNA strand is its finish, much estem a shoelace. Because the lose, flapy ends of your shoelaces are much more likely to get frayed, shoemakers protect them with those protective plastic caps. Telomeres, sequences of proteins that live on either stop of your DNA strands, serve honest like those protective caps on your shoelaces: They replicate diferently and are therefore not prone to the same kind of aflict as the rest of a DNA strand. However, every time DNA replicates, these telomeres get “worn down” objective a dinky bit. When telomeres become to short, they signal to the cel that “it’s time to die.” When scientists observed that mice with longer telomeres lived longer and had les DNA pain than shorter-telomered mice, they theorized that stimulating the production of telomerase–the enzyme that lengthens telomeres–might be the secret to slowing or reversing aging.
    Of course, nothing is simple when it comes to the multiplex proces of aging: Exces telomerase is also linked with cancer. It has even yet to be proved if telomere length is a cause of aging or merely a byproduct of it. love other theories of the rot cause of aging, telomere theory is a definite “maybe.”
    Botom line.
    And so it goes. There are many more theories of aging–some are quite briliant and ad considerably to our understanding of why and how we age, but none suced in fuly explaining the causes and mechanisms of aging.
    The folowing is an excerpt from The Science Technology of Growing Young by Sergey Young, with permision by BenBela Boks.
    The Science Technology of Growing Young

    Author:Sergey Young
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